Signal Transduction by Reactive Oxygen and Nitrogen Species: Pathways and Chemical Principles

Henry Jay Forman, Jon Fukuto and Martine Torres "Research is to see what everybody else has seen and to think what nobody else has thought. " -- Albert Szent-Gyorgyi Several years ago, one of us put together a book that dealt with various aspects of oxidative stress and introduced the conc...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Άλλοι συγγραφείς: Forman, Henry Jay (Επιμελητής έκδοσης), Fukuto, Jon (Επιμελητής έκδοσης), Torres, Martine (Επιμελητής έκδοσης)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: Dordrecht : Springer Netherlands, 2003.
Θέματα:
Διαθέσιμο Online:Full Text via HEAL-Link
Πίνακας περιεχομένων:
  • Redox State and Redox Environment in Biology
  • Sulfur and Selenium Catalysis as Paradigms for Redox Regulations
  • Thiols of Thioredoxin and Glutaredoxin in Redox Signaling
  • Reactivity and Diffusivity of Nitrogen Oxides in Mammalian Biology
  • Bacterial Peroxiredoxins
  • The Nox Enzymes and the Regulated Generation of Reactive Oxygen Species
  • NO Synthesis and NOS Regulation
  • S-nitrosothiols in Cell Signaling
  • Hydrogen Peroxide as Intracellular Messenger: Identification of Protein Tyrosine Phosphatases and Pten as H2O2 Target
  • 4-Hydroxynonenal Signaling
  • Ceramide Signaling under Oxidative Stress
  • Map Kinases in Redox Signaling
  • Activation of Prostaglandin Biosynthesis: Peroxynitrite vs Hydroperoxides
  • Phylogenetic Conservation of the Nrf2-Keap1 Signaling System
  • The NO-cytochrome C Oxidase Signaling Pathway; Mechanisms and Biological Implications
  • The Concept of Compartmentalization in Signaling by Reactive Oxygen Species
  • Role of Mitochondrial Oxygen and Nitrogen Reactive Species in Signaling
  • Redox Regulation of Gene Expression: Transcriptional Induction of Heme Oxygenase-1
  • Apoptosis Mechanisms Initiated by Oxidative Stress
  • Reactive Oxygen and Nitrogen Species in the Production of Congenital Malformations by Known Teratogenic Agents and Maternal Conditions.