Acute Neuronal Injury The Role of Excitotoxic Programmed Cell Death Mechanisms /

Acute Neuronal Injury The Role of Excitotoxic Programmed Cell Death Mechanisms /

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This book is the result of a convergence of scientific information regarding mechanisms that produce acute nerve cell death in the brain. Although seemingly disparate, stroke, brain and spinal cord trauma, coma from a low serum glucose concentration (hypoglycemia), and prolonged epileptic seizures h...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Άλλοι συγγραφείς: Fujikawa, Denson G. (Επιμελητής έκδοσης)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: Boston, MA : Springer US, 2010.
Θέματα:
Medicine.
Neurosciences.
Neurology.
Pathology.
Psychiatry.
Cell biology.
Neurobiology.
Biomedicine.
Cell Biology.
Διαθέσιμο Online:Full Text via HEAL-Link
Πίνακας περιεχομένων:
  • Caspase-Independent Programmed Cell Death: General Considerations
  • Caspase-Independent Cell Death Mechanisms in Simple Animal Models
  • Programmed Necrosis: A “New” Cell Death Outcome for Injured Adult Neurons?
  • Age-Dependence of Neuronal Apoptosis and of Caspase Activation
  • Excitotoxic Programmed Cell Death Involves Caspase-Independent Mechanisms
  • Focal Cerebral Ischemia
  • Significant Role of Apoptosis-Inducing Factor (AIF) for Brain Damage Following Focal Cerebral Ischemia
  • The Role of Poly(ADP-Ribose) Polymerase-1 (PARP-1) Activation in Focal Cerebral Ischemia
  • Transient Global Ischemia
  • Transient Global Cerebral Ischemia Produces Morphologically Necrotic, Not Apoptotic Neurons
  • Apoptosis-Inducing Factor Translocation to Nuclei After Transient Global Ischemia
  • Role of µ-Calpain I and Lysosomal Cathepsins in Hippocampal Neuronal Necrosis After Transient Global Ischemia in Primates
  • Traumatic Central Nervous System (CNS) Injury
  • Mitochondrial Damage in Traumatic CNS Injury
  • Programmed Neuronal Cell Death Mechanisms in CNS Injury
  • Hypoglycemic Neuronal Death
  • Hypoglycemic Brain Damage
  • Hypoglycemic Neuronal Death
  • Seizure-Induced Neuronal Death
  • Tumor Suppressor p53: A Multifunctional Protein Implicated in Seizure-Induced Neuronal Cell Death
  • DNA Damage and Repair in the Brain: Implications for Seizure-Induced Neuronal Injury, Endangerment, and Neuroprotection
  • Activation of Caspase-Independent Programmed Pathways in Seizure-Induced Neuronal Necrosis.

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