Chemokine Receptors and NeuroAIDS Beyond Co-Receptor Function and Links to Other Neuropathologies /

Chemokines and their receptors are being recognized as an integral component of the nervous system implicated in fundamental aspects of development and homeostasis, such as neurotransmission, proliferation, differentiation, and neuronal-glial communication. Thus, their involvement in HIV neuropathol...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Άλλοι συγγραφείς: Meucci, Olimpia (Επιμελητής έκδοσης)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: New York, NY : Springer New York : Imprint: Springer, 2010.
Θέματα:
Διαθέσιμο Online:Full Text via HEAL-Link
Πίνακας περιεχομένων:
  • Section I
  • HIV Neuroinvasion: Early Events, Late Manifestations
  • HIV Co-receptors: The Brain Perspective
  • HIV Infection and the PNS
  • HIV Latency and Reactivation: Role in Neuropathogenesis
  • HIV Coreceptors and Their Roles in Leukocyte Trafficking During Neuroinflammatory Diseases
  • Section II
  • Chemokine Proteolytic Processing in HIV Infection: Neurotoxic and Neuroimmune Consequences
  • Chemokines and Chemokine Receptors in the Brain
  • Chemokine Signaling in the Nervous System and Its Role in Development and Neuropathology
  • Modulation of Neuronal Cell Cycle Proteins by Chemokine Receptors and Its Role in the Survival of Postmitotic Neurons
  • Chemokines and Primary Brain Tumors
  • Chemokines as Neuromodulators: Regulation of Glutamatergic Transmission by CXCR4-Mediated Glutamate Release From Astrocytes
  • Role of CX3CL1 in Synaptic Activity and Neuroprotection
  • Section III
  • Interaction Between Opioid and Chemokine Receptors in Immune Cells: Implications for HIV Infection
  • Chronic Morphine’s Role on Innate Immunity, Bacterial Susceptibility and Implications in Wound Healing
  • Opioids, Astroglial Chemokines, Microglial Reactivity, and Neuronal Injury in HIV-1 Encephalitis
  • Regulation of Neuronal Chemokine Receptor CXCR4 by ?-Opioid Agonists and Its Involvement in NeuroAIDS.