Kernicterus
Kernicterus is an excellent example of a metabolic encephalopathy. It clearly produces a biochemical lesion early when bilirubin first enters the brain. If initial staining can be minimized or reversed, permanent damage can be avoided. This takes strict attention to detail, and a keen ability for ti...
Κύριος συγγραφέας: | |
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Συγγραφή απο Οργανισμό/Αρχή: | |
Μορφή: | Ηλεκτρονική πηγή Ηλ. βιβλίο |
Γλώσσα: | English |
Έκδοση: |
Totowa, NJ :
Humana Press : Imprint: Humana Press,
2011.
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Σειρά: | Contemporary Clinical Neuroscience
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Θέματα: | |
Διαθέσιμο Online: | Full Text via HEAL-Link |
Πίνακας περιεχομένων:
- Dedication page
- Preface
- Acknowledgement
- Key words.-Chapter 1 Prologue: Hyperbilirubinemia, kernicterus, and world health concerns.-Chapter 2 History of bilirubin.-Chapter 3 Biochemistry and physiology of bilirubin
- Chapter 4 Prematurity
- Chapter 5 Erythroblastosis fetalis
- Chapter 6 Gunn rats
- Chapter 7 Crigler-Najjar Syndrome
- Chapter 8 Neuropathology of kernicterus
- Chapter 9 Bilirubin and energy metabolism
- Chapter 10 Bilirubin and other biochemical changes
- Chapter 11 Jaundice and breast milk
- Chapter 12 Jaundice and malaria
- Chapter 13 Jaundice and congenital pyloric stenosis
- Chapter 14 Phototherapy for hyperbilirubinemia
- Chapter 15 Non phototherapy treatment
- Chapter 16 Hyperbilirubinemia revisited
- Chapter 17 Auditory brainstem response
- Chapter 18 Progressive familial intrahepatic cholestasis
- Chapter 19 Kernicterus in older children and adults
- Chapter 20 Cerebral palsy and counseling
- Chapter 21 Neurological sequelae from jaundice
- Chapter 22 Neurobehavioral teratology
- Chapter 23 Gene therapy for hyperbilirubinemia
- Chapter 24 Epilog: Kernicterus: comments and future directions
- Appendix
- References
- Index.