Kernicterus

Kernicterus is an excellent example of a metabolic encephalopathy. It clearly produces a biochemical lesion early when bilirubin first enters the brain. If initial staining can be minimized or reversed, permanent damage can be avoided. This takes strict attention to detail, and a keen ability for ti...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Κύριος συγγραφέας: McCandless, David W. (Συγγραφέας)
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: Totowa, NJ : Humana Press : Imprint: Humana Press, 2011.
Σειρά:Contemporary Clinical Neuroscience
Θέματα:
Διαθέσιμο Online:Full Text via HEAL-Link
Πίνακας περιεχομένων:
  • Dedication page
  • Preface
  • Acknowledgement
  • Key words.-Chapter 1 Prologue: Hyperbilirubinemia, kernicterus, and world health concerns.-Chapter 2 History of bilirubin.-Chapter 3 Biochemistry and physiology of bilirubin
  • Chapter 4 Prematurity
  • Chapter 5 Erythroblastosis fetalis
  • Chapter 6 Gunn rats
  • Chapter 7 Crigler-Najjar Syndrome
  • Chapter 8 Neuropathology of kernicterus
  • Chapter 9 Bilirubin and energy metabolism
  • Chapter 10 Bilirubin and other biochemical changes
  • Chapter 11 Jaundice and breast milk
  • Chapter 12 Jaundice and malaria
  • Chapter 13 Jaundice and congenital pyloric stenosis
  • Chapter 14 Phototherapy for hyperbilirubinemia
  • Chapter 15 Non phototherapy treatment
  • Chapter 16 Hyperbilirubinemia revisited
  • Chapter 17 Auditory brainstem response
  • Chapter 18 Progressive familial intrahepatic cholestasis
  • Chapter 19 Kernicterus in older children and adults
  • Chapter 20 Cerebral palsy and counseling
  • Chapter 21 Neurological sequelae from jaundice
  • Chapter 22 Neurobehavioral teratology
  • Chapter 23 Gene therapy for hyperbilirubinemia
  • Chapter 24 Epilog: Kernicterus: comments and future directions
  • Appendix
  • References
  • Index.