Cardiac Remodeling Molecular Mechanisms /
The main objective of Cardiac Remodeling: Molecular Mechanisms is to summarize the major research advances in molecular, biochemical and translational aspects of cardiac remodeling over the last 2 to 3 decades under one cover and touch on future directions. It provides a high profile and valuable pu...
| Συγγραφή απο Οργανισμό/Αρχή: | |
|---|---|
| Άλλοι συγγραφείς: | , |
| Μορφή: | Ηλεκτρονική πηγή Ηλ. βιβλίο |
| Γλώσσα: | English |
| Έκδοση: |
New York, NY :
Springer New York : Imprint: Springer,
2013.
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| Σειρά: | Advances in Biochemistry in Health and Disease ;
5 |
| Θέματα: | |
| Διαθέσιμο Online: | Full Text via HEAL-Link |
Πίνακας περιεχομένων:
- Part I: Molecular mechanisms of remodeling in pressure and volume overload hypertrophy and ehart failure
- β-adrenergic receptor signaling pathway in heart failure
- Remodeling of potassium channels in cardiac hypertrophy
- Role of gender in Ca2+ cycling and cardiac remodeling due to heart failure
- The failing heart: is it an inefficient engine or an engine out of fuel?
- Regulation of cardiac hypertrophic remodeling by the USP15/SLIM1 pathway
- Role of galectin-3 pathways in the pathogenesis of cardiac remodeling and heart failure
- A mitochondriocentric pathway to cardiomyocyte necrosis: an upstream molecular mechanism in myocardial fibrosis
- The ACE2/Ang (1-7) pathway in cardiac remodeling due to pressure-overload
- Local actions of natriuretic peptides and nitric oxide in cardiac remodelling: Implications for therapy
- Modulating G protein-coupled receptors to effect reverse cardiac remodeling
- Role of inflammation and matrix proteinases in cardiac remodeling following stress and injury
- Role of chymase in matrix and myocardial remodeling due to mitral regurgitation. Implications for therapy
- Cardiac remodeling due to aortic regurgitation and mitral regurgitation
- Reducing oxidative stress and manipulating molecular signaling events using resveratrol as a therapy for pathological cardiac hypertrophy
- Angiogenesis, arteriogenesis, and mitochondrial dysfunction
- Part II: Molecular mechanisms of remodeling after myocardial injury and infarction
- Subcellular remodeling and cardiac dysfunction due to ischemia-reperfusion injury
- Role of microRNAs in cardiac hypertrophy and post-infarction remodeling
- Negative regulators of inflammation as endogenous protective mechanisms in post-infarction remodeling
- TLR-dependent pathways and Akt/mTOR/P70S6K pathways in cardiac remodeling after myocardial infarction
- The STAT3 pathway and downstream mechanisms in cardiac remodeling: Friend or foe
- The role of growth differentiation factor 5 in cardiac repair post myocardial infarction
- Extracellular matrix biomarkers of adverse remodeling after myocardial infarction
- Oxidative stress in cardiac repair and remodeling: Molecular pathways and therapeutic strategies
- Role of SPARC in cardiac extracellular matrix remodeling after myocardial infarction
- Tissue inhibitor of matrix metalloproteinases in the pathogenesis of heart failure syndromes
- Intracellular matrix remodeling and cardiac function in ischemia-reperfusion injury
- Aging and markers of adverse remodeling after myocardial infarction
- Optimizing stem cell therapy for cardiac repair following a myocardial infarction
- Regulation of fibrosis after myocardial infarction. Implications for ventricular remodeling
- The ACE2/Ang-(1-7) pathway in cardiac fibroblasts as a potential target for cardiac remodeling.
