Opiate Receptors and Antagonists From Bench to Clinic /

The evolution in our understanding of Opioid receptors and their subtypes is intimately linked to the development of new pharmacological treatments for diseases and disorders as diverse as addiction, self-injurious behavior, pain, cancer, inflammation, eating disorders, traumatic injury, pruritis an...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Άλλοι συγγραφείς: Dean, Reginald L. (Επιμελητής έκδοσης), Bilsky, Edward J. (Επιμελητής έκδοσης), Negus, S. Stevens (Επιμελητής έκδοσης)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: Totowa, NJ : Humana Press, 2009.
Σειρά:Contemporary Neuroscience
Θέματα:
Διαθέσιμο Online:Full Text via HEAL-Link
Πίνακας περιεχομένων:
  • Opioid Receptors
  • Ultra-Low-Dose Opioid Antagonists Enhance Opioid Analgesia and Reduce Tolerance
  • Upregulation of Opioid Receptors
  • Imaging Human Brain Opioid Receptors: Applications to Substance Use Disorders
  • Opioid Receptor Antagonist-Mediated Signaling in the Immune System
  • Opioid Antagonists: Chemistry and Pharmacology
  • The Chemistry and Pharmacology of ?-Opioid Antagonists
  • Medicinal Chemistry of Kappa Opioid Receptor Antagonists
  • The Chemistry and Pharmacology of Delta Opioid Antagonists
  • Novel Opioid Antagonists with Mixed/Dual Selectivity
  • Experimental Utility and Clinical Potential of Irreversible Opioid Antagonists
  • Methylnaltrexone: A Peripherally Acting Opioid Antagonist
  • Substance Abuse
  • Opioid Antagonist Effects in Animal Models Related to Opioid Abuse: Drug Discrimination and Drug Self-Administration
  • Naltrexone for Initiation and Maintenance of Opiate Abstinence
  • Ultra-Low-Dose Naltrexone Decreases Dependence and Addictive Properties of Opioids
  • Can a Combination Formulation Containing a Neutral Opiate Antagonist Decrease the Abuse of ?-Agonist Opiates
  • Effects of Opioid Antagonists on the Abuse-Related Effects of Psychomotor Stimulants and Nicotine
  • Potential Use of Opioid Antagonists in the Treatment of Marijuana Abuse and Dependence
  • Naltrexone in Smoking Cessation: A Review of the Literature and Future Directions
  • Alcohol and Ingestive Behaviors
  • Opioid Antagonists and Ethanol's Ability to Reinforce Intake of Alcoholic Beverages: Preclinical Studies
  • Clinical Use of Opioid Antagonists in the Treatment of Alcohol Dependence
  • Preclinical Effects of Opioid Antagonists on Feeding and Appetite
  • CNS Opiate Systems and Eating Disorders
  • Behavioral Disorders
  • Potential Utility of Kappa Ligands in the Treatment of Mood Disorders
  • Opioid Antagonists in the Treatment of Pathological Gambling and Kleptomania
  • Efficacy of Opioid Antagonists in Attentuating Self-Injurious Behavior
  • Pharmacotherapeutic Effects of Opioid Antagonists in Alcohol-Abusing Patients with Schizophrenia
  • Medical Indications
  • Current Issues in the Use of Opioid Antagonists (Naltrexone for Opiate Abuse: A Re-Educational Tool as Well as an Effective Drug)
  • Emergency Room Use of Opioid Antagonists in Drug Intoxication and Overdose
  • Kappa-Opioid Antagonists as Pruritogenic Agents
  • Clinical Effect of Opioid Antagonists on Clinical Pruritus
  • Effects of Opioid Antagonists on l-DOPA-Induced Dyskinesia in Parkinson's Disease
  • Endocrine Effects of Opioid Antagonists
  • Opioid Antagonists in Traumatic Shock: Animal and Human Studies
  • The Efficacy of Opioid Antagonists Against Heatstroke-Induced Ischemia and Injury in Rats
  • A Review of the Opioid System in Cancer Patients and Preliminary Results of Opioid Antagonists in the Treatment of Human Neoplasms
  • Nonclinical Pharmacology of VIVITROL®: A Monthly Injectable Naltrexone for the Treatment of Alcohol Dependence
  • The Development of Sustained-Release Naltrexone and Clinical Use in Treating Opiate Dependence
  • The Development of ProNeura Technology for the Treatment of Addictions
  • Development of Opioid Transdermal Delivery Systems
  • Intranasal Naloxone for Treatment of Opioid Overdose.