Transforming Growth Factor-β in Cancer Therapy, Volume I Basic and Clinical Biology /
Transforming Growth Factor-ß in Cancer Therapy, Volumes 1 and 2, provide a compendium of findings about the role of transforming growth factor-ß (TGF-ß) in cancer treatment and therapy. The first volume, Basic and Clinical Biology, is divided into three parts. Part I presents basic concepts of trans...
Κύριος συγγραφέας: | |
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Συγγραφή απο Οργανισμό/Αρχή: | |
Μορφή: | Ηλεκτρονική πηγή Ηλ. βιβλίο |
Γλώσσα: | English |
Έκδοση: |
Totowa, NJ :
Humana Press,
2008.
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Σειρά: | Cancer Drug Discovery and Development
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Θέματα: | |
Διαθέσιμο Online: | Full Text via HEAL-Link |
Πίνακας περιεχομένων:
- Basic Concepts of TGF-? Signaling in Normal Physiology and Cancer Pathobiology
- Component Hardware for Transforming Growth Factor-? Signal Transduction: TGF-? Ligands, TGF-? Receptors, and Smads
- TGF-? Signaling in Homeostasis and Cancer
- TGF-? Availability: Latent TGF-? and Latent TGF-? Binding Proteins
- Biological Functions of Latent TGF-?-Binding Proteins and Activation of TGF-?
- Regulators of Smad2/3 Transcription and Phosphorylation
- Interaction of Smad4 and Embryonic Liver Fodrin-?-spectrin in Hyperplasia, Neoplasia, and Tumor Suppression
- Regulation of Smad Activity by Phosphorylation
- Nuclear Targeting of TGF-?-Activated Smads in Normal and Tumor Biology
- Ski, SnoN, and Akt as Negative Regulators of Smad Activity: Balancing Cell Death and Cell Survival
- Transforming Growth Factor-? in Cancer Therapy: Smurfs in TGF-? Signaling and Regulation of Bone Homeostasis and Cancer
- Involvement of km23 Dynein Light Chains in TGF ? Signaling
- TGF-? Signaling in Fibroblastic Cells and Oncogenesis
- Mechanisms of TGF-?-Induced Apoptosis in Cancer Cells
- Mechanisms of Cell Cycle Regulation by TGF-? Disabled in Cancer
- Transforming Growth Factor-? in Cancer Therapy
- TGF-? and Smad Signaling in Transcriptome Reprogramming During EMT
- Functional Interactions Between MMPs and TGF-? in Normal and Tumor Tissue
- Cooperation Between TGF-? and Wnt Pathway Components in Regulating Mesenchymal Stem Cell Function
- Interaction of Oncogenic Ras and TGF?1 Signaling in Cancer: Lessons From the Multistage Skin Carcinogenesis Model
- TGF-? Superfamily Members in Normal and Tumor Biology
- Proteomics Analysis of TGF? Superfamily Members
- Mullerian Inhibiting Substance: A Potential Nontoxic, Naturally Occurring Therapeutic Agent for Certain Human Cancers
- Activins, Inhibins, and Bone Morphogenetic Proteins as Modulators and Biomarkers of Prostate Cancer Progression
- Bone Morphogenic Proteins and Angiogenesis in Developing Tumors
- Histone Methyltransferase and Smad Interactions in BMP Signaling
- Endoglin (CD105): A Strong Candidate for Immunologic Targeting of Tumor Neovasculature in Human Malignancies
- Activins and Inhibins in Cancer Progression
- Bone Morphogenetic Proteins/Growth Differentiation Factors and Smad Activation in Ovarian Granulosa Cells and Carcinoma
- Regulation of the Transforming Growth Factor-? Superfamily by Betaglycan
- Uterine Sensitization Associated Gene-1: A Bone Morphogenetic Protein Antagonist with a Role in Kidney
- Role of Betaig-h3 Gene in Carcinogenesis
- Activins and the Liver
- Myostatin Signaling in Normal and Tumor Cells
- TGF-? in Inflammation and Fibrosis
- Triterpenoids, Vitamin D, and TGF-?/Smad Signaling in Inflammation and Cancer
- Thrombospondin-Dependent Activation of Latent TGF-? in Fibrosis and Disease
- TGF-? in Pulmonary Fibrosis
- The Role of TGF-? in Bleomycin Induced Pulmonary Fibrosis
- Smads in the Fibrotic Response: Findings in the Smad3 Knockout Mouse
- TGF-? Signaling in Renal Fibrosis and Inflammation: Gene Therapy Using Ultrasound-Microbubble-Mediated Inducible Smad7
- Transforming Growth Factor-? Peptide Signaling in Pulmonary Development, Bronchopulmonary Dysplasia, Fibrosis, and Emphysema
- The Role of TGF-? in Radiation and Chemotherapy Induced Pulmonary Fibrosis: Inhibition of TGF-? as a Novel Therapeutic Strategy
- Negative Regulation of the TGF-? Family Signal Pathway by Inhibitory Smads and Their Involvement in Cancer and Fibrosis
- TGF-?-Related Antifibrotic Strategies in the Eye
- Transforming Growth Factor-? in Cutaneous Tissue Repair
- Topical Application of TGF-?1 Peptide Inhibitors for the Therapy of Skin Fibrosis
- Overexpressed Truncated TGF-? Type II Receptor Inhibits Fibrotic Behavior of Keloid Fibroblasts In Vitro and Experimental Scar Formation In Vivo.