Advances in multiple Sclerosis and Experimental Demyelinating Diseases
"There is a need for a paradigm shift in our thinking about the pathogenesis of multiple sclerosis." Challenging Charcot’s hypothesis that inflammatory response is the primary contributor to demyelination, Dr. Rodriguez and colleagues take a fresh, bold look at the causes and possible trea...
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| Format: | Electronic eBook |
| Language: | English |
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Berlin, Heidelberg :
Springer Berlin Heidelberg,
2008.
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| Series: | Current Topics in Microbiology and Immunology,
318 |
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| Online Access: | Full Text via HEAL-Link |
Table of Contents:
- Benign Multiple Sclerosis: A Distinct Clinical Entity with Therapeutic Implications
- Pathological Heterogeneity of Idiopathic Central Nervous System Inflammatory Demyelinating Disorders
- Multiple Sclerosis Genetics
- Imaging of Remyelination and Neuronal Health
- Immunological Aspects of Axon Injury in Multiple Sclerosis
- The Multiple Sclerosis Degradome: Enzymatic Cascades in Development and Progression of Central Nervous System Inflammatory Disease
- Genetic Analysis of CNS Remyelination
- Remyelination in Experimental Models of Toxin-Induced Demyelination
- Remyelination-Promoting Human IgMs: Developing a Therapeutic Reagent for Demyelinating Disease
- Neuroimaging of Demyelination and Remyelination Models
- Hormonal Influences in Multiple Sclerosis
- Statins and Demyelination
- Role of Uric Acid in Multiple Sclerosis
- Neuromyelitis Optica: Clinical Syndrome and the NMO-IgG Autoantibody Marker.
