Tumor Dormancy, Quiescence, and Senescence, Volume 1 Aging, Cancer, and Noncancer Pathologies /
With a particular emphasis on tumor dormancy in breast, lung, prostate, and liver cancers, as well as in melanoma, this first volume of a new Springer series focuses on the interrelationship between biological processes of aging and tumors—both dormant and quiescent. With detail supplied by numerous...
| Συγγραφή απο Οργανισμό/Αρχή: | |
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| Άλλοι συγγραφείς: | |
| Μορφή: | Ηλεκτρονική πηγή Ηλ. βιβλίο |
| Γλώσσα: | English |
| Έκδοση: |
Dordrecht :
Springer Netherlands : Imprint: Springer,
2013.
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| Σειρά: | Tumor Dormancy and Cellular Quiescence and Senescence, Aging, Cancer, and Noncancer Pathologies ;
1 |
| Θέματα: | |
| Διαθέσιμο Online: | Full Text via HEAL-Link |
Πίνακας περιεχομένων:
- Preface.-I. Tumor dormancy
- Is tumor dormancy clinically relevant?- Microenvironmental influence on breast cancer Dormancy and metastasis
- Determination of breast cancer dormancy: analysis of Circulating free dna using snp 6.0 arrays
- Clonogenicity of cultured prostate cancer cells is controlled by dormancy : significance and comparison with cell culture models of breast cancer cell dormancy
- Dormancy and metastasis of melanoma cells to lymph Nodes, lung, and liver
- Late recurrence is a sign of melanoma dormancy: need of Life-long follow-up of elanoma patients. II. Quiescence
- Hematopoietic stem cell quiescence and long term Maintenance: role of scl/tal1
- Regulation of muscle stem cell quiescent and Undifferentiated state: roles of hesr1 and hesr3 genes
- The kinase mirk/dyrk1b mediates a reversible quiescent State in a subset of ovarian, pancreatic, and colon cancers. III. Cellular senescence
- Stress -induced senescence: molecular pathways.-11 Accumulation of reactive oxygen species and induction Of premature senescence: role of ddb2
- P21 mediates senescence by a mechanism involving accumulation of reactive oxygen species
- Role of micrornase and zeb1 downmodulation in oxidative Stress-induced apoptosis and senescence.- Hypoxic response in senescent brain is impaired: possible Contribution to neurodegeneration.-Enhancing reprogramming to pluripotency by controlling senescence
- Histone deacetylase inhibitor induces replicative Senescence of mesenchymal stem cells
- Senescence arrest of endopolyploid cells renders Senescence into one mechanism for positive tumorigenesis
- The two faces of senescence-associated epigenetic Alterations: tumor suppressors and oncogenic drivers.- Chemotherapy- and radiation-induced accelerated senescence: implications for treatment response, tumor progression, and cancer survivorship
- Suppression of cellular senescence in glioblastoma: role Of src homology domain-containing phosphatase 2
- Chemotherapy of malignant pleural mesothelioma induces both senescence and apoptosis
- Microrna as a modulator of cell proliferation and Senescence: role in lung cancer cells
- Role of senescence induction in cancer therapy
- Cellular senescence limits the extent of fibrosis Following liver damage
- Formation of secretory senescent cells in prostate tumors: the role of androgen receptor activity and cell cycle regulation.-Index .
