Modulation of Oxidative Stress in Heart Disease

This book highlights the multifaceted roles of Reactive Oxygen Species (ROS) in modulating normal cellular and molecular mechanisms during the development of different types of heart disease. Each chapter in the book deals with the role that altered redox homeostasis plays in the pathophysiology of...

Πλήρης περιγραφή

Λεπτομέρειες βιβλιογραφικής εγγραφής
Συγγραφή απο Οργανισμό/Αρχή: SpringerLink (Online service)
Άλλοι συγγραφείς: Chakraborti, Sajal (Επιμελητής έκδοσης, http://id.loc.gov/vocabulary/relators/edt), Dhalla, Naranjan S. (Επιμελητής έκδοσης, http://id.loc.gov/vocabulary/relators/edt), Dikshit, Madhu (Επιμελητής έκδοσης, http://id.loc.gov/vocabulary/relators/edt), Ganguly, Nirmal K. (Επιμελητής έκδοσης, http://id.loc.gov/vocabulary/relators/edt)
Μορφή: Ηλεκτρονική πηγή Ηλ. βιβλίο
Γλώσσα:English
Έκδοση: Singapore : Springer Singapore : Imprint: Springer, 2019.
Έκδοση:1st ed. 2019.
Θέματα:
Διαθέσιμο Online:Full Text via HEAL-Link
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245 1 0 |a Modulation of Oxidative Stress in Heart Disease  |h [electronic resource] /  |c edited by Sajal Chakraborti, Naranjan S. Dhalla, Madhu Dikshit, Nirmal K. Ganguly. 
250 |a 1st ed. 2019. 
264 1 |a Singapore :  |b Springer Singapore :  |b Imprint: Springer,  |c 2019. 
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520 |a This book highlights the multifaceted roles of Reactive Oxygen Species (ROS) in modulating normal cellular and molecular mechanisms during the development of different types of heart disease. Each chapter in the book deals with the role that altered redox homeostasis plays in the pathophysiology of heart disease. In addition, the book explains how reactive oxidant species interact with their targets and provides novel strategies for attenuating oxidative stress-induced types of heart disease. The book not only covers ROS-induced response in heart disease at the cellular level, but also demonstrates that an imbalance of redox states has its roots in our genes, and explains the ways gene expression is regulated. In turn, it reviews potential sources of ROS, their pathological effects on the heart, and potential sites for therapeutic interventions. 
650 0 |a Oxidative stress. 
650 0 |a Cardiovascular system. 
650 0 |a Cardiology. 
650 0 |a Cell physiology. 
650 0 |a Biochemistry. 
650 0 |a Biomedical engineering. 
650 1 4 |a Oxidative Stress.  |0 http://scigraph.springernature.com/things/product-market-codes/L16070 
650 2 4 |a Cardiovascular Biology.  |0 http://scigraph.springernature.com/things/product-market-codes/L16090 
650 2 4 |a Cardiology.  |0 http://scigraph.springernature.com/things/product-market-codes/H33037 
650 2 4 |a Cell Physiology.  |0 http://scigraph.springernature.com/things/product-market-codes/L33010 
650 2 4 |a Biochemistry, general.  |0 http://scigraph.springernature.com/things/product-market-codes/L14005 
650 2 4 |a Biomedical Engineering/Biotechnology.  |0 http://scigraph.springernature.com/things/product-market-codes/B24000 
700 1 |a Chakraborti, Sajal.  |e editor.  |4 edt  |4 http://id.loc.gov/vocabulary/relators/edt 
700 1 |a Dhalla, Naranjan S.  |e editor.  |4 edt  |4 http://id.loc.gov/vocabulary/relators/edt 
700 1 |a Dikshit, Madhu.  |e editor.  |4 edt  |4 http://id.loc.gov/vocabulary/relators/edt 
700 1 |a Ganguly, Nirmal K.  |e editor.  |4 edt  |4 http://id.loc.gov/vocabulary/relators/edt 
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