9781003032762_10.4324_9781003032762-30.pdf

Debates about the etiology of addiction have a long history and continue to the present day. In contemporary societies, the brain disease model of addiction (BDMA) continues to receive strong support, in particular, from US agencies such as the National Institute on Drug Abuse (NIDA) and the America...

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Έκδοση: Taylor & Francis 2022
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spelling oapen-20.500.12657-522002022-01-06T02:47:26Z Chapter 26 Addiction treatment providers’ engagements with the Brain Disease Model of Addiction Barnett, Anthony Savic, Michael Pickersgill, Martyn O’Brien, Kerry Lubman, Dan I. Carter, Adrian brain disease model; addiction; treatment bic Book Industry Communication::M Medicine::MM Other branches of medicine::MMZ Therapy & therapeutics Debates about the etiology of addiction have a long history and continue to the present day. In contemporary societies, the brain disease model of addiction (BDMA) continues to receive strong support, in particular, from US agencies such as the National Institute on Drug Abuse (NIDA) and the American Society of Addiction Medicine. Today, there continues to be a significant investment in addiction neuroscience research globally. However, the views of addiction treatment providers about the BDMA, and its clinical impact, are often ignored when debates led by public health researchers and neuroscientists dominate discourse about the neurobiology of addiction. In this chapter, we start by providing a brief history of the biomedicalization of addiction. Moving beyond the question of ‘Is addiction a brain disease, or not?’, we summarize providers’ views about the BDMA and its impact on clinical practice. Drawing on recent critical drug studies scholarship, we critique how a simplistic, linear ‘bench to bedside’ model of addiction neuroscience translation elides the role treatment providers play in translating neuroscience. Finally, we consider the effects of how the enactment of addiction as a brain disease within policy impacts treatment, and how addiction might be enacted in other ways in future policy frameworks. 2022-01-05T10:46:46Z 2022-01-05T10:46:46Z 2022 chapter 9780367470043 9780367470067 https://library.oapen.org/handle/20.500.12657/52200 eng application/pdf Attribution-NonCommercial-NoDerivatives 4.0 International 9781003032762_10.4324_9781003032762-30.pdf Taylor & Francis Evaluating the Brain Disease Model of Addiction 10.4324/9781003032762-30 10.4324/9781003032762-30 7b3c7b10-5b1e-40b3-860e-c6dd5197f0bb 4d2fe20d-1506-4bfa-ad11-bd6e873f11e3 9780367470043 9780367470067 13 open access
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language English
description Debates about the etiology of addiction have a long history and continue to the present day. In contemporary societies, the brain disease model of addiction (BDMA) continues to receive strong support, in particular, from US agencies such as the National Institute on Drug Abuse (NIDA) and the American Society of Addiction Medicine. Today, there continues to be a significant investment in addiction neuroscience research globally. However, the views of addiction treatment providers about the BDMA, and its clinical impact, are often ignored when debates led by public health researchers and neuroscientists dominate discourse about the neurobiology of addiction. In this chapter, we start by providing a brief history of the biomedicalization of addiction. Moving beyond the question of ‘Is addiction a brain disease, or not?’, we summarize providers’ views about the BDMA and its impact on clinical practice. Drawing on recent critical drug studies scholarship, we critique how a simplistic, linear ‘bench to bedside’ model of addiction neuroscience translation elides the role treatment providers play in translating neuroscience. Finally, we consider the effects of how the enactment of addiction as a brain disease within policy impacts treatment, and how addiction might be enacted in other ways in future policy frameworks.
title 9781003032762_10.4324_9781003032762-30.pdf
spellingShingle 9781003032762_10.4324_9781003032762-30.pdf
title_short 9781003032762_10.4324_9781003032762-30.pdf
title_full 9781003032762_10.4324_9781003032762-30.pdf
title_fullStr 9781003032762_10.4324_9781003032762-30.pdf
title_full_unstemmed 9781003032762_10.4324_9781003032762-30.pdf
title_sort 9781003032762_10.4324_9781003032762-30.pdf
publisher Taylor & Francis
publishDate 2022
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